RESUMEN
OBJECTIVE: The purpose of this study was to evaluate the in vitro shearing force performance of orthodontic attachments using two self-etching primers (SEPs): iBOND and G-Bond. DESIGN: In vitro, laboratory study. MATERIAL AND METHODS: One hundred and eighty human molars were randomly divided into four groups and again into three sub-groups with 15 teeth each. Teeth were bonded with a stainless steel button (GAC International,Bohemia, NY, USA) using Transbond XT adhesive composite. The bonding agents were iBOND, G-Bond, Transbond Plus SEP and Transbond XT primer. Shearing force tests were carried out immediately, and at 24 hours and 3 months using a universal testing machine. Force to debond (N) and Adhesive Remnant Index (ARI) scores were evaluated and compared. RESULTS: Transbond XT primer required a higher immediate (P<0·05)force to debond when compared to the Transbond Plus SEP, iBOND and G-Bond.After 24 hours, mean force to debond for Transbond XT primer and Transbond Plus SEP showed significant increases. At 3 months, all four bonding agents demonstrated force levels to debond that were not significantly different from one another. Furthermore, comparison of ARI scores indicated a significant difference between the groups at all time points. CONCLUSIONS: iBOND and G-Bond may well sufficiently with stand the alignment and occlusal forces imparted by light archwires during immediate archwire tie-in and over the initial levelling and alignment phase.
Asunto(s)
Recubrimiento Dental Adhesivo , Grabado Dental/métodos , Análisis del Estrés Dental , Cementos de Resina/química , Análisis de Varianza , Resinas Compuestas , Humanos , Estimación de Kaplan-Meier , Ensayo de Materiales , Metacrilatos , Diente Molar , Aparatos Ortodóncicos , Resistencia al CorteRESUMEN
Treponema denticola and its major outer sheath protein (Msp) induce actin reorganization in fibroblasts. We adapted a barbed-end labeling/imaging assay to monitor Msp-induced subcortical actin filament assembly in neutrophils and fibroblasts. Msp, at an actin-reorganizing concentration, inhibited migration of these dissimilar cell types, whose cytoskeletal functions in locomotion and phagocytosis are crucial for immunity and healing of peripheral infections.