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Knee dislocation remains a devastating injury with many complications. It necessitates prompt diagnosis, reduction if needed, and emergent repair of any vascular injury. Serial physical examinations and frequent use of arteriograms are necessary to avoid late vascular complications. Many authors are concerned that normal pulses, normal Doppler signals, and normal ABIs have preceded late ischemia and documented intimal tear, demonstrated by arteriography. More recently, other authors have challenged the gold standard of mandatory arteriography by describing studies in which physical examination was 100% accurate in diagnosing patients without operative vascular injury. If pedal pulses, Doppler signals, or ABIs are asymmetric before or after reduction then either immediate operative exploration or arteriography should be performed. If the initial physical examination is normal, serial examinations are used in the hospital to check for late artery thrombosis. Opponents of mandatory arteriography point to a 5% false-negative rate, high cost, and an 8% complication rate, such as contrast allergy, pseudoaneurysm, local hematoma, and arteriovenous fistula. Today a consensus is that repair and reconstruction of the PCL and posterolateral corner injuries are the primary concerns in the multiple-ligament injured knee after dislocation. The ACL may be repaired later if instability persists, but some investigators believe it should not be repaired acutely, thereby avoiding increased surgical trauma and possible stiffness. Recently one of the goals of ligamentous repair and reconstruction has been to provide stability with the least invasive surgical technique to avoid postoperative stiffness. Recent treatments have focused on early arthroscopic-assisted allograft reconstruction of the ACL and PCL. Allograft provides a less invasive means of graft support than autograft. Early, limited range of motion in a brace helps to maintain flexion and extension.

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OBJECTIVE: We outline impingement entities, describe the history and physical examination, and provide an overview of treatment beyond that routinely used in glenohumeral and scapulothoracic dysfunction. BACKGROUND: In the athlete, pain and dysfunction due to excessive overhead use or abnormal positioning of the shoulder is common and can result from multiple etiologies, including impingement syndromes. Primary, secondary, internal, and coracoid impingement have all been described. DESCRIPTION: These entities will be discussed, including pathology, evaluation, and treatment. CLINICAL ADVANTAGES: Incorporating a systematic evaluation and treatment of impingement syndromes optimizes care for the patient with shoulder pain.

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This investigation documented the locations of endoscopically applied T-Fix suture devices (Acufex Microsurgical, Mansfield, MA) placed in six fresh-frozen cadaveric knees (age, 60 to 72 years) in relationship to the joint capsule, and adjacent neurovascular and musculotendinous structures. Five T-Fix devices were placed in the posterior meniscal regions at approximately 20 degree intervals. Gross dissection enabled T-Fix bar and suture placement identification. Fifty total devices were placed (23 medially and 27 laterally). Lateral: None of the devices penetrated more superficially than the deepest capsular layer (layer III). Six of the 27 devices placed at the posterior horn of the lateral meniscus pierced the popliteus tendon. None of the bars pierced the lateral collateral ligament (layer III). All devices placed at the posterolateral knee were outside the arcuate ligament (layer III) but inside the fabellofibular ligament (layer II). Medial: Seven of the 23 devices pierced the deep medial collateral ligament (MCL, layer III), and 4 pierced the superficial MCL (layer II). Three devices pierced the sartorius tendon (layer I) and one pierced the gracilis tendon (layer II). None of the medial devices created a plicating effect on the posterior capsule. None of the devices were placed near neurovascular structures. Devices placed within the posterior meniscal horns had a > or =1.5-cm buffer zone from the popliteal neurovascular bundle. Most bars (36 of 50) were anchored to the capsular layer (layer III) after piercing the meniscocapsular junction (layer II). T-Fix devices simulating arthroscopic all-inside meniscal repair provided well-positioned, solid suture anchorage through the junction with no neurovascular involvement. Care needs to be taken when placing lateral (popliteus muscle) and medial (gracilis, sartorius tendons and superficial MCL) devices to avoid possible soft tissue tenodesis.

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The authors report the arthroscopic finding of articular cartilage "dimpling" when a probe is placed onto the discrete chondral area involved over the geographic bone bruise incurred during traumatic anterior cruciate ligament disruption. As we develop an understanding of the pathology and sequelae of this osteochondral injury, this finding may be useful to document injury extent and possibly guide treatment including weight-bearing status and rehabilitation.

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In an experimental evaluation with 14 paired cadaveric scapulae we found that the transacromial arthroscopy portal, used occasionally in the repair of superior labral lesions, will reduce the structural integrity of the acromion to approximately 60% (range 25% to 85%) of its original strength, thereby placing it at increased risk of fracture. These studies provide baseline biomechanical information and suggest that limited shoulder activity is indicated after use of this portal.

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