RESUMEN

Mechanism tobacco favours arteriosclerosis aren't clear. Hyperglycemia in smokers would be help factor. On value relationship between tobacco, fasting plasma glucose and hemoglobin A1c in a coal-miner working population, made up of 1,379 males, 38 years old average. We find meaning difference in hemoglobin A1c and fasting plasma glucose values between smokers and non-smokers. Hemoglobin A1c are more elevated in smokers while glycemia are more elevated in non-smokers. We rapport some reason over these divergent results. Hemoglobin A1c elevated in smokers suggest that the tobacco has negative influence on glucose metabolism.

Asunto(s)

Metabolismo de los Hidratos de Carbono , Fumar/metabolismo , Adulto , Arteriosclerosis/epidemiología , Glucemia/análisis , Hemoglobina Glucada/análisis , Humanos , Masculino , Persona de Mediana Edad , Minería , Nicotina/farmacocinética , Factores de Riesgo