RESUMEN
Group B beta-hemolytic streptococcus (GBS) has emerged as a major cause of neonatal infections. Serious GBS infections in adults occur most frequently when predisposing illness is present. Recurrent erysipelas caused by GBS is reported in a woman who had chronic lymphedema after a radical hysterectomy and radiation therapy. Type III GBS was identified from cultures of vulvar vesicles, vaginal mucosa, and blood. Prophylactic penicillin V potassium therapy prevented further episodes of erysipelas. Group B beta-hemolytic streptococcus should be considered a possible cause of erysipelas or cellulitis.
Asunto(s)
Erisipela/etiología , Abdomen/patología , Erisipela/tratamiento farmacológico , Erisipela/patología , Femenino , Humanos , Dermatosis de la Pierna/etiología , Dermatosis de la Pierna/patología , Persona de Mediana Edad , Penicilina V/uso terapéutico , Recurrencia , Streptococcus agalactiae , Vulva/patologíaAsunto(s)
Alopecia/complicaciones , Dermatosis Facial/complicaciones , Neoplasias Faciales/complicaciones , Mucinosis Folicular/complicaciones , Micosis Fungoide/complicaciones , Adulto , Dermatosis Facial/patología , Neoplasias Faciales/patología , Femenino , Humanos , Mucinosis Folicular/patología , Mucinosis Folicular/terapia , Micosis Fungoide/patología , Micosis Fungoide/terapiaRESUMEN
The glucagonoma syndrome is characterized by dermatitis, stomatitis, elevated serum glucagon levels, abnormal glucose tolerance, weight loss, and anemia--all in association with a glucagon-secreting alpha-cell tumor of the pancreas. A review of 21 cases showed strikingly similar features. A generalized, symmetrical dermatitis initially appeared to be asteatotic or eczematous over the perineum, buttocks, and lower extremities. Gradually, a more characteristic migratory necrolytic erythema with transient bulla formation and erosions developed in intertriginous and dependent areas. Histologically, the most specific features included necrolysis of the upper epidermis, with liquefaction necrosis of the granular cell layer and subcorneal clefting or blister formation. The dermatologist is often first to examine such patients; early recognition of this syndrome with prompt surgical removal of the primary pancreatic lesion may afford cure of the neoplasm.